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Free heme and the polymerization of sickle cell hemoglobin

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At this point, we have previously shown that IGF-I is a main character in restoring mitochondrial dysfunction during aging by increasing mitochondrial membrane potential, reducing oxygen consumption, and increasing ATP synthesis what in turn minimize the c-cytochrome release to the cytoplasm and subsequently promote neural survival by decreasing caspase-induced apoptosis [55,56], in agreement with in vitro reported works from others [242-244]

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Sikiri et al., Dig