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We hypothesized that L-carnitine, when administered alongside DMARDs, would reduce disease activity and modulate key molecular markers including STAT3 and TGF-1

Mechanistically, inhibition of the PI3K/AKT/mTOR axis resulted in increased expression of pro-apoptotic proteins caspase-3, caspase-9, and Bax, while reducing the expression of the anti-apoptotic protein Bcl-2, thereby accelerating apoptotic events in LCSCs

Uric acid stimulates hepatic steatosis either directly or by activating NLRP3 inflammasomes

Some studies suggest that high levels of L-carnitine may raise the long-term risks of cardiovascular diseases, though more research is necessary

Numerous studies have suggested a decrease in atherosclerotic disease risk [heart disease, our #1 killer] in vegan and vegetarian individuals compared to omnivores