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Drug targeting by long- circulating liposomal glucocorticosteroids increases therapeutic efficacy in a model of multiple sclerosis

AFB1- exo -8,9-epoxide is then able to bind to guanine residues in DNA thus confering mutagenic properties

Elevated blood TMAO levels cause an imbalance in cholesterol homeostasis which leads to influenced macrophages, which in turn causes foam cell development and atherosclerotic plaques [68, 69, 71]

Another suggests that a combination of smoking and alcohol can decrease glutathione activity (17)

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