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This resulted in desensitization to MPTP-induced permeability transition, thereby suppressing the expression of mitochondrial apoptosis signals, including initiator Cyto C, mediator caspase-9, and effector caspase-3

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The observed accumulation of LDH and ALT in vascular medium supernatants compared to supernatants from the static hepatic chamber could be attributed to increased permeability upon cytotoxicity and subsequent accumulation of toxicity markers in the vascular perfusion (Supplementary Fig