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Ipamorelin and Hexarelin instead act on the ghrelin receptor (GHS-R1a) in the pituitary and hypothalamus, triggering GH release through a separate pathway

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Based on these new insights, a theory has been proposed that explains the pathogenesis of MASLD through processes that exceed the normal metabolic capacity of the liver, leading to the excessive accumulation of toxic lipids, hepatocellular stress, injury, and cell death [40]