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However, upon entering the reperfusion phase, oxidative stress, inflammatory responses, and mitochondrial damage intensify, leading to the activation of death receptors such as TNF-R and Fas

For clarity, not all pathways, co-substrates or products are shown and some intermediates are omitted (indicated by dotted lines)

To date, direct evidence for the effect of AMPK on renal cell ferroptosis under diabetic conditions is not sufficient