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The first (classical) pathway is as follows: under normal conditions Nrf2 is localized in cytoplasm and associated with its inhibitor KEAP1 (Kelch-like ECH (Nrf2)-associated protein-1), which stimulates Nrf2 ubiquitinylation and its subsequent proteasomal degradation

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Palmatine reduced Heparin-binding epidermal growth factor-like growth factor (HB-EGF) and p-EGFR/EGFR levels, suppressing HAG progression