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Because the therapeutic potential of supplemental coenzyme Q 10 is limited to its capacity to restore electron transfer in a defective mitochondrial respiratory chain and/or to increase antioxidant defense, patients with secondary coenzyme Q 10 deficiency may fail to respond to supplementation (see Disease Treatment)

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To address this, Ciulli and co-workers [256] introduced ferrocene as a structurally dynamic linker, capitalizing on its inherent chameleonicity to develop a class of degraders termed FerroTACs (Fig