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It has a longer duration in the body due to its slow release and extended half-life, often requiring less frequent dosing

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EA at LI11 and ST36 reduced infarct volume by attenuating the overactivation of Iba-1 and ED1-positive microglia in the peri-infract sensorimotor cortex, decreasing the level of TNF-, IL-1, and IL-6 in serum, and preventing the nucleus translocation of NF-B p65 and the expression of p38 mitogen-activated protein kinase (p38 MAPK) and myeloid differentiation primary response gene 88 (MyD88) in the peri-infract sensorimotor cortex

CNS Neurosci Ther 30(12):e70147 Mai Z, Sun H, Yang F, Du M, Cheng X, Chen H, Sun B, Wen J, Wang X, Chen T (2023) Bad is essential for Bcl-xL-enhanced Bax shuttling between mitochondria and cytosol

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