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This process generates an abundance of ROS, ion dysregulation (including but not limited to iron ions), glutamate-mediated excitotoxicity, and immune-related neurotoxicity

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Taken together, the present study demonstrates that CAE ameliorates cognitive impairments through a synergistic mechanism that involves (1) the restoration of cholinergic function, (2) the enhancement of intrinsic antioxidant defenses, (3) the inhibition of apoptotic processes, and (4) the activation of neurotrophic signaling pathways

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The non-thiol sulfur amino acids methionine and cystathionine were also increased in urine, suggesting loss of cysteine precursors, possibly related to the effect of BSO in kidney